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Troponin T binds the troponin factors to tropomyosin. Troponin I inhibits the interaction of myosin with actin. Troponin C is made up of the binding web-sites for that Ca2+ that assists to initiate contraction. [one]
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Scientific tests in people receiving chemotherapeutic agents with identified cardiotoxicity have revealed that those who build elevated troponin stages in the absence of ischemia are at an elevated long term risk of creating remaining ventricular failure and cardiomyopathy. [24]
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